All Publications
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XWAS: a toolset for genetic data analysis and association studies of the X chromosome. Journal of Heredity. .
106(5):6 (2015)
X-inactivation informs variance-based testing for X-linked association of a quantitative trait. BMC Genomics. .
16:241 (2015)
Who does what: Taking measures. Statistical and Process Models for Cognitive Neuroscience and Aging. .
:163 (2007)
The utility of ancient human DNA for improving allele age estimates, with implications for demographic models and tests of natural selection.. Journal of human evolution. .
(2014) Abstract
Strong Constraint on Human Genes Escaping X-Inactivation Is Modulated by their Expression Level and Breadth in Both Sexes. Molecular Biology and Evolution. .
33(2):384-393 (2015) Abstract
Spikes that count: rethinking spikiness in neurally embedded systems. Neurocomputing. .
58:303–311 (2004) Abstract
Solving a delayed response task with spiking and McCulloch-Pitts agents. Advances in Artificial Life. .
:199–208 (2003) Abstract
Sex-averaged recombination and mutation rates on the X chromosome: a comment on Labuda et al. Am. J. Hum. Genet.. .
86:978–980;authorreply980-981 (2010) Abstract
Selection for translation efficiency on synonymous polymorphisms in recent human evolution. Genome Biol Evol. .
3:749–761 (2011) Abstract
Reply to Just et al.: Mitochondrial DNA heteroplasmy could be reliably detected with massively parallel sequencing technologies.. Proceedings of the National Academy of Sciences of the United States of America. .
111(43):E4548-50 (2014)
Recent explosive human population growth has resulted in an excess of rare genetic variants. Science. .
336:740–743 (2012) Abstract
Rare LPL gene variants attenuate triglyceride reduction and HDL cholesterol increase in response to fenofibric acid therapy in individuals with mixed dyslipidemia.. Atherosclerosis. .
234(2):249-253 (2014) Abstract
Quantitative analysis of genetic and neuronal multi-perturbation experiments. PLoS Comput. Biol.. .
1(6):e64 (2005) Abstract
Principal component analysis characterizes shared pathogenetics from genome-wide association studies.. PLoS computational biology. .
10(9):e1003820 (2014) Abstract
Predicting signatures of "synthetic associations" and "natural associations" from empirical patterns of human genetic variation. PLoS Comput. Biol.. .
8:e1002600 (2012) Abstract
Positive Selection on a Regulatory Insertion-Deletion Polymorphism in FADS2 Influences Apparent Endogenous Synthesis of Arachidonic Acid.. Molecular Biology and Evolution. .
33(7):1726-39 (2016) Abstract
Population growth inflates the per-individual number of deleterious mutations and reduces their mean effect.. Genetics. .
195(3):969-78 (2013) Abstract
Population Genomic Analysis of 962 Whole Genome Sequences of Humans Reveals Natural Selection in Non-Coding Regions. PLoS ONE. .
10:e0121644 (2015) Abstract
Population genetic inference from personal genome data: impact of ancestry and admixture on human genomic variation. Am. J. Hum. Genet.. .
91:660–671 (2012) Abstract
NRE: a tool for exploring neutral loci in the human genome. BMC Bioinformatics. .
13(1):301 (2012) Abstract
Neutral genomic regions refine models of recent rapid human population growth.. Proceedings of the National Academy of Sciences of the United States of America. .
111(2):757-62 (2014) Abstract
Neurocontroller analysis via evolutionary network minimization. Artif. Life. .
12:435–448 (2006) Abstract
Neural processing of counting in evolved spiking and McCulloch-Pitts agents. Artif. Life. .
12:1–16 (2006) Abstract
Mitochondrial DNA variants correlate with symptoms in myalgic encephalomyelitis/chronic fatigue syndrome.. Journal of translational medicine. .
14(1):19 (2016) Abstract